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31. Follow - up brain CT revealed a hemorrhagic infarction over right temporal lobe.

32. EI values were computed in mesial and lateral structures of the temporal lobe in a group of 17 patients with mesial temporal lobe epilepsy (MTLE).

33. This artery supplies portions of the MESENCEPHALON (see also BRAIN STEM INFARCTIONS) and thalamus, inferomedial TEMPORAL LOBE, and medial OCCIPITAL LOBE.

34. Objective To explore whether medial temporal lobe (MTL) atrophy would possibly predict the cognitive decline in the progression of Alzheimer's disease(AD).

35. Objective : To explore effect of AEEG, CT and MRI in diagnosis of temporal lobe epilepsy.

36. Objective:To evaluate the value of proton magnetic resonance spectroscopy (1H-MRS)in the localization diagnosis (LD) of temporal lobe epilepsy (TLE).

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37. The expression of NGB in the surgically resected brain tissues(hippocampus and temporal lobe)in 7 cases with medically intractable epilepsy was observed using immunohistochemistry.

38. Here we investigate whether the amygdala, a temporal lobe structure, is affected by ALS.

39. Methods 24 patients with intractable temporal lobe epilepsy were operated via modified pterional approach with good microsurgical technique and the epileptogenic focuses were removed.

40. Objective To study the diagnosis and surgical operation in the treatment of temporal lobe epilepsy with hippocampal atrophia .

41. Objective To discuss the diagnosis and surgical treatment of temporal lobe epilepsy.

42. The hospital's CAT scanner showed that two jagged pieces of Herring's skull had been driven deep into the left temporal lobe of her brain.

43. This prospective study compared preoperative quantitative hippocampal MR imaging volumetry and MRS imaging in 33 patients with intractable medial temporal lobe epilepsy (TLE).

44. The hemorrhages seen here in the temporal lobe are due to Herpes simplex virus infection.

45. Methods: Adopting through the pterion the temporal lobe and frontal lobe were divided with vessels protected, then the tumors were removed piece by piece under the microscopy.

46. Conclusions: 1 There were a few brain neuron injuries of premature rats induced by status epilepticus, mainly in hippocampal CA3, following by hippocampus CA1, dentate gyrus, temporal lobe.

47. Results Gliosis was observed, to different extent, in the brain tissues from patients with refractory temporal lobe epilepsy, so were neuronophagia phenomenon and neuronal degeneration.

48. Of most concern to Dr. Schneider , however, was her left temporal lobe.

49. There is the frontal lobe, easy to remember, it's part in front, the parietal lobe, the occipital lobe, and the temporal lobe.

50. Objective To study the surgery of temporal lobe epilepsy caused by mass lesions or hippocampus sclerosis.

51. Volume of hippocampal formation was measured by MRI in 39 patients with temporal lobe epilepsy and in 14 patients with frontal lobe epilepsy.

52. Objective To discuss the characteristics of subgroup neu ron loss of hippocampus sclerosis (HS) in temporal lobe epilepsy (TLE).

53. A few years later German neurologist Carl Wernicke identified a second language center farther back, in the brain's left temporal lobe.

54. Conclusions:The alerting network function of the controls maybe correlated with the encephalic region of right occipital lobe, cerebellum, right frontal lobe, brain stem and temporal lobe.

55. The mass extends inferiorly into the infratemporal fossa and left retropharyngeal space. There is compression of anteromedial left temporal lobe, but no significant edema.

56. Results The kinds of temporal lobe seizures included simple partial, complex partial and generalized tonic clonic. Complex partial seizure was the most common one.

57. This sort of processing happens in the primary auditory cortex, both left and right, of the temporal lobe.

58. AIM To observe the stability of the injection of kainic acid into lateral ventricle to build chronic temporal lobe epilepsy rat model.

59. Mixed high - valence facial expressions are processed within temporal lobe visual cortex, thalamus, and amygdalohippocampal complex.

60. To try to identify the critical structures during epileptogenesis, we used the lithium-pilocarpine model that reproduces most clinical and neuropathological features of temporal lobe epilepsy (TLE).